Alzheimer's Disease and Related Disorders Annual - 2001 by Jeffrey L Cummings, Serge Gauthier

By Jeffrey L Cummings, Serge Gauthier

Alzheimer's affliction is a seriously researched zone with medical trials carrying on with and new healing recommendations and items to evaluate. The editors and participants tackle modern themes during this zone in a few comprehensively referenced chapters, designed to stimulate dialogue on the place present therapy is heading. individuals to this annual are all the world over well known specialists of their subspecialties according to scientific event and medical proof for the therapy of this illness.

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The ‘exclusionary’ approach Traditionally, computed tomography (CT) and magnetic resonance imaging (MRI) were used to exclude other illnesses that were potentially amenable to (surgical) treatment. The yield of such a procedure probably is less than 1%. Recently Farina et al10 performed CT in 513 patients referred to a memory clinic of whom 362 were found to be demented. 2%) a potential reversible cause of dementia was detected. However, CT revealed no findings that had not been discovered clinically.

4 illustrates autopsy specimens of brain from patients who had fatal CAA-related hemorrhage. 18 Awareness of CAA as a likely etiology for lobar hemorrhage is also important for the surgical pathologist; CAA can be detected readily in brain parenchyma adjacent to an evacuated hematoma,50,51 provided the examiner’s index of suspicion is appropriately high and the patient’s ‘profile’ fits that of one likely to present with this type of stroke. Patients are usually in their 70s or 80s, and (see above) may be demented prior to the occurrence of stroke, although often they are not.

Neuroimaging Clin N America 1992; 2:153–169. 44. Coria F, Rubio I. Cerebral amyloid angiopathies. Neuropathol Appl Neurobiol 1996; 22:216–227. 45. Fountain NB, Eberhard DA. Primary angiitis of the central nervous system associated with cerebral amyloid angiopathy: report of two cases and review of the literature. Neurology 1996; 46:190–197. 46. Ohshima T, Endo T, Nukui H, et al. Cerebral amyloid angiopathy as a cause of subarachnoid hemorrhage. Stroke 1990; 21:480–483. 47. Gray F, Dubas F, Roullet E, Escourolle R.

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